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This Concept Map, created with IHMC CmapTools, has information related to: blanks_Viral path, evade by Antibodies function as opsonins and sticking viruses to phagocytes., evade by down regulating several host proteins involved in attaching viral epitopes to MHC-I molecules, Pathogenicity of Animal Viruses damaging infected host cells depleting the host cell of cellular materials essential for life or normal function, example congenital rubella syndrome, inducing adjacent host cells to fuse together forming giant multinucleated cells or syncytias results in, evade by producing microRNAs that blocks apoptosis of virus-infected cells, evade by inhibiting proteasomal activity, Pathogenicity of Animal Viruses damaging infected host cells, evade by blocking the TAP transport of peptides into the endoplasmic reticulum, evade by blocking the formation of MHC-I molecules by the infected cell, results in vasodilation and increased mucous secretion, Pathogenicity of Animal Viruses immune defenses against viruses Antibodies function as opsonins and sticking viruses to phagocytes., Pathogenicity of Animal Viruses damaging infected host cells, Pathogenicity of Animal Viruses damaging infected host cells causing cytolysis of the infected host cell, Type-I interferons produced during innate immunity block viral replication. evade by, Pathogenicity of Animal Viruses immune defenses against viruses NK cells recognize infected cells displaying stressed-induced proteins and not displaying MHC-I molecules on their surface and kill these cells., Type-I interferons produced during innate immunity block viral replication. evade by, Pathogenicity of Animal Viruses damaging infected host cells inhibiting normal host cell DNA, RNA, or protein synthesis, Pathogenicity of Animal Viruses damaging infected host cells, Pathogenicity of Animal Viruses immune defenses against viruses
